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Crohn's Disease- Pathophysiology and Conventional and Alternative Treatment Options

Crohn's Disease

Table 1: Subcategories of Crohn's Disease

Table 2: Signs and Symptoms of Crohn's Disease and Ulcerative Colitis

Risk Factors



    The Genetic Component of Crohn's Disease

    Stress in the Etiology of Crohn's Disease

    Microbial Factors

    Inflammation/Immune Response

    Intestinal Permeability

    Other Abnormalities Contributing to the Etiopathogenesis of Crohn's Disease

Conventional Treatment of Crohn's Disease

Table 5: Conventional Medications and their Mechanisms in Crohn's Disease

Nutrient Deficiences in Crohn's Disease

Table 6: nutrient Deficiencies Associated with Crohn's Disease

Dietary Interventions in Crohn's Disease

Table 7: Diet Therapies Compared to Steroid Medications in Crohn's Disease

Probiotics in the Treatment of Crohn's Disease

Fatty Acids for the Treatment of Crohn's Disease

Table 8: A Summary of Omega-3 Fatty Acid Studies in Crohn's Disease


N-acetyl Glucosamine

Remicade Increasing Risk of Cancer

Botanicals in the Treatment of Crohn's Disease

Dehydroepiandrosterone (DHEA)

Potential Sequelae of Crohn's Disease


Table 5. Conventional Medications and their Mechanisms in Crohn’s Disease

Medication Mechanisms of Action
Aminosalicylates Anti-inflammatory (slow-release topically to small bowel)
Corticosteroids Anti-inflammatory; immunosuppressive
Other Immunosuppressive Agents Suppress the immune response in Crohn's
Antibiotics Damage cell wall of pathogenic agents; reduce bacterial load
TNF-alpha Monoclonal Antibodies TNF-alpha neutralization and apoptosis
Anti-sense Agents Modulate lymphocyte migration to gut mucosa
Anti-interleukins Decrease inflammation by inhibiting inflammatory cytokines
Mitogen-activated Protein Kinase Inhibitors Indirectly inhibit TNF-alpha
Somatatropin Decreases intestinal permeability; decreases mesenteric fat; increases amino acid and electrolyte absorption in intestine
Sargramostim Possible immunostimulation of neutrophils

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